Soluble RAGE levels have been identified as hypothetical markers of CVD, but, in this regard, there are sparse and conflicting data in the literature.
The purpose of this review was to examine all the available information on this issue with a view to clarifying or at least highlighting the points that are still weak, especially from the point of clinical view.
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In this sense, hyperglycemia is an important contributor to the onset of cardiovascular complications.
Among the biochemical alterations characteristic of hyperglycemia, the factors involved in causing atherosclerotic disease include the formation of AGEs, an increased polyol pathway flux and hexosamine pathway flux, and protein kinase C activation .
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All of these molecular mechanisms reflect a single hyperglycemia-induced process of superoxide overproduction by the mitochondrial electron transport chain.
Hyperglycemia and an increased oxidative stress  thus lead to tissue damage via common pathways.
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